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Figure 1.
Lipid metabolism disorders in mother-child liver in response to malnutrition during late gestation. BHBA, β-hydroxybutyrate; NEFAs, non-esterified fatty acids; LPA, lysophosphatidic acid; LPC, lysophosphatidylcholine; LPE, lysophosphatidylethanolamine; TGs, triglycerides; VLDL, very low-density lipoprotein; PA, phosphatidic acid; PC, phosphatidylcholine; PE, phosphatidylethanolamine.
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Figure 2.
Differentially expressed genes involved in lipid metabolism in mother-child liver in response to malnutrition during late gestation. MG, monoglyceride; NEFAs, non-esterified fatty acids; TGs, triglycerides.
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Figure 3.
Maternal malnutrition-induced lipid metabolic disorders in fetal liver inhibit fetal development. NEFAs, non-esterified fatty acids; LPA, lysophosphatidic acid; LPC, lysophosphatidylcholine; LPE, lysophosphatidylethanolamine.
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Figure 4.
Maternal malnutrition affects fetal liver lipid metabolism and oxidative stress. BHBA, beta-hydroxybutyrate; BMI, body mass index; NEFAs, non-esterified fatty acids; PLs, phospholipids; TC, cholesterol; TGs, triglycerides.
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